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Epithelial- and Endothelial- to Mesenchymal Transition: from Cardiovascular Development to Disease.
Cryoinjury as a myocardial infarction model for the study of cardiac regeneration in the zebrafish.
The Osr1 and Osr2 genes act in the pronephric anlage downstream of retinoic acid signaling and upstream of Wnt2b to maintain pectoral fin development.
Matrigel-embedded 3D culture of hepatoma-derived cell lines as a hepatocyte-like polarized system to study hepatitis C virus cycle.
Inhibition of transforming growth factor-activated kinase 1 (TAK1) blocks and reverses epithelial to mesenchymal transition of mesothelial cells.
Influence of bicarbonate-low-GDPs peritoneal dialysis fluid (Bicavera®) on in vitro and ex vivo epithelial-to-mesenchymal transition of mesothelial cells
GSK3, Snail and adhesion molecule regulation by Cyclosporine A in renal tubular cells.
TWEAK (tumor necrosis factor-like weak inducer of apoptosis) activates CXCL16 expression during renal tubulointerstitial inflammation.
Pharmacogenetics of tacrolimus REDINREN study group. Association between a common KCNJ11 polymorphism (rs5219) and new-onset posttransplant diabetes in patients treated with Tacrolimus.
Pharmacological modulation of peritoneal injury induced by dialysis fluids: is it an option?
HSP27/HSPB1 as an adaptive podocyte antiapoptotic protein activated by high glucose and angiotensin II.
THE INFLUENCE OF INITIAL PERITONEAL TRANSPORT CHARACTERISTICS, INFLAMMATION, AND HIGH GLUCOSE EXPOSURE ON PROGNOSIS FOR PERITONEAL MEMBRANE FUNCTION.
Cost comparison between haemodialysis and peritoneal dialysis outsourcing agreements.
Hypoxia inducible factor 1-alpha (HIF-1 alpha) is induced during reperfusion after renal ischemia and is critical for proximal tubule cell survival.
Medical therapy with tranexamic acid in autosomal dominant polycystic kidney disease patients with severe haematuria.
Thyroid function tests in acute kidney injury.
Cinacalcet improves control of secondary hyperparathyroidism in peritoneal dialysis: a multicenter study.
TWO-IN-ONE PROTOCOL: SIMULTANEOUS SMALL-PORE AND ULTRASMALL-PORE PERITONEAL TRANSPORT QUANTIFICATION.
Effect of self-administered intraperitoneal bemiparin on peritoneal transport and ultrafiltration capacity in peritoneal dialysis patients with membrane dysfunction. A randomized, multi-centre open clinical trial.
Cost-effectiveness analysis of the Spanish renal replacement therapy program.
Inhibition of focal adhesion kinase prevents experimental lung fibrosis and myofibroblast formation.
Critical role of hydrogen peroxide signaling in the sequential activation of p38 MAPK and eNOS in laminar shear stress.
The non-canonical NOTCH ligand DLK1 exhibits a novel vascular role as a strong inhibitor of angiogenesis.
Intracellular redox equilibrium is essential for the constitutive expression of AP-1 dependent genes in resting cells: Studies on TGF-β1 regulation.
New losartan-hydrocaffeic acid hybrids as antihypertensive-antioxidant dual drugs: Ester, amide and amine linkers.
Start-up of a clinical simple processing, storage and management platform: organisation and development of the REDinREN Biobank.
A polymeric nanomedicine diminishes inflammatory events in renal tubular cells.
Angiotensin II Contributes to Renal Fibrosis Independently of Notch Pathway Activation
Follistatin-Like 3 Mediates Paracrine Fibroblast Activation by Cardiomyocytes
Understanding cardiovascular disease: a journey through the genome (and what we found there).
Pan-epicardial lineage tracing reveals that epicardium derived cells give rise to myofibroblasts and perivascular cells during zebrafish heart regeneration
Epithelial-to-mesenchymal and endothelial-to-mesenchymal transition: from cardiovascular development to disease.
LOXL4 is induced by TGF-β1 through Smad and JunB/Fra2 and contributes to vascular matrix remodeling.2388-401.
ILK conditional deletion in adult animals increases cyclic gmp-dependent vasorelaxation
Tamoxifen Ameliorates Peritoneal Membrane Damage by Blocking Mesothelial to Mesenchymal Transition in Peritoneal Dialysis.
Functional Relevance of the Switch of VEGF Receptors/Co-Receptors during Peritoneal Dialysis-Induced Mesothelial to Mesenchymal Transition.
Are the mesothelial-to-mesechymal transition, sclerotic peritonitis syndromes and encapsulating peritoneal sclerosis part of the same process?
The mesothelial to mesenchymal transition a pathogenic and therapeutic key for peritoneal membrane failure.
Encapsulating peritoneal sclerosis in peritoneal dialysis. A review and European initiative for approaching a serious and rare disease
The C-terminal module IV of connective tissue growth factor is a novel immune modulator of the Th17 response.
Heartbeat-driven pericardiac fluid forces contribute to epicardium morphogenesis
The profibrotic role of endothelin-1: Is the door still open for the treatment of fibrotic diseases?
S-glutathionylation: relevance in diabetes and potential role as a biomarker.
The alternative heart: impact of alternative splicing in heart disease.
Carcinoma-associated fibroblasts derive from mesothelial cells via mesothelial-to-mesenchymal transition in peritoneal metastasis.
Induction of the calcineurin variant CnAβ1 after myocardial infarction reduces post-infarction ventricular remodelling by promoting infarct vascularization.
FineSplice, enhanced splice junction detection and quantification: a novel pipeline based on the assessment of diverse RNA-Seq alignment solutions.
The Epicardium in the Embryonic and Adult Zebrafish.
TWEAK promotes peritoneal inflammation.
Paricalcitol Reduces Peritoneal Fibrosis in Mice through the Activation of Regulatory T Cells and Reduction in IL-17 Production.
PERITONEAL RESTING WITH HEPARINIZED LAVAGE REVERSES PERITONEAL TYPE I MEMBRANE FAILURE. A COMPARATIVE STUDY OF THE RESTING EFFECTS ON NORMAL MEMBRANES.
Peritoneal dialysis can be an option for dominant polycystic kidney disease: an observational study
Histological and clinical findings in patients with post-transplantation and classical encapsulating peritoneal sclerosis: a European multicenter study.
Macrophages and recently identified forms of cell death.
A Pathogenetic Role for Endothelin-1 in Peritoneal Dialysis-Associated Fibrosis.
IL-17A is a novel player in dialysis-induced peritoneal damage
Mesenchymal Conversion of Mesothelial Cells is a Key Event inthe Pathophysiology of the Peritoneum During Peritoneal Dialysis
Angiotensin II, via angiotensin receptor type 1/nuclear factor-κB activation, causes a synergistic effect on interleukin-1-β-induced inflammatory responses in cultured mesangial cells.
Targeting of γ-glutamyl-cysteine ligase by miR-433 reduces glutathione biosynthesis and promotes TGF-β-dependent fibrogenesis.
The C-Terminal Module IV of Connective Tissue Growth Factor, Through EGFR/Nox1 Signaling, Activates the NF-κB Pathway and Proinflammatory Factors in Vascular Smooth Muscle Cells.
Gremlin activates the Smad pathway linked to epithelial mesenchymal transdifferentiation in cultured tubular epithelial cells.
Tubular overexpression of gremlin induces renal damage susceptibility in mice
Translational study of the Notch pathway in hypertensive nephropathy
Integrin-linked kinase plays a key role in the regulation of angiotensin II-induced renal inflammation.
Osteoprotegerin in exosome-like vesicles from human cultured tubular cells and urine.
Connective tissue growth factor is a new ligand of epidermal growth factor receptor.
Use of echocardiography reveals reestablishment of ventricular pumping efficiency and partial ventricular wall motion recovery upon ventricular cryoinjury in the zebrafish.
Transcriptional response to cardiac injury in the zebrafish: systematic identification of genes with highly concordant activity across in vivo models.
Biocompatibility Reduces Inflammation-Induced Apoptosis in Mesothelial Cells Exposed to Peritoneal Dialysis Fluid.
Elevated expression levels of lysyl oxidases protect against aortic aneurysm progression in Marfan syndrome
T Helper 17/Regulatory T Cell Balance and Experimental Models of Peritoneal Dialysis-Induced Damage
TGF-Beta Blockade Increases Renal Inflammation Caused by the C-Terminal Module of the CCN2
miR-9-5p suppresses pro-fibrogenic transformation of fibroblasts and prevents organ fibrosis by targeting NOX4 and TGFBR2.
Antioxidant responses and cellular adjustments to oxidative stress.
Matrix cross-linking lysyl oxidases are induced in response to myocardial infarction and promote cardiac dysfunction.
Origin and evolution of lysyl oxidases.
Workshop on cardiovascular extracellular matrix in health and disease in Baeza, Spain
Guidelines for translational research in heart failure.
Telomerase Is Essential for Zebrafish Heart Regeneration.
Biocompatible Dialysis Solutions Preserve Peritoneal Mesothelial Cell and Vessel Wall Integrity. A Case-Control Study on Human Biopsies.
The Mesothelial Origin of Carcinoma Associated-Fibroblasts in Peritoneal Metastasis.